the atm protein is a single high molecular weight protein predominantly confined to the nucleus of human
fibroblasts but is present in both nuclear and microsomal fractions from human lymphoblast cells and peripheral
blood lymphocytes atm protein levels and localization remain constant throughout all stages of the cell cycle
truncated atm protein was not detected in lymphoblasts from ataxia telangiectasia patients homozygous
for mutations leading to premature protein termination exposure of normal human cells to gamma irradiation and the
radiomimetic drug neocarzinostatin had no effect on atm protein levels in contrast to a noted rise in p53 levels
over the same time interval these findings are consistent with a role for the atm protein in ensuring the fidelity
of dna repair and cell cycle regulation following genome damage

will be punctuated as follows:

The ATM protein is a single, high-molecular-weight protein predominantly confined to the nucleus of human
fibroblasts, but is present in both nuclear and microsomal fractions from human lymphoblast cells and peripheral
blood lymphocytes. ATM protein levels and localization remain constant throughout all stages of the cell cycle.
Truncated ATM protein was not detected in lymphoblasts from ataxia-telangiectasia-patients homozygous
for mutations leading to premature protein termination. Exposure of normal human cells to gamma-irradiation and the
radiomimetic drug neocarzinostatin had no effect on ATM protein levels, in contrast to a noted rise in p53 levels
over the same time interval. These findings are consistent with a role for the ATM protein in ensuring the fidelity
of DNA repair and cell-cycle regulation following genome damage.

How to use it in your code:

python

import torch
import numpy as np
from transformers import DistilBertTokenizerFast, DistilBertForTokenClassification

checkpoint = "unikei/distilbert-base-re-punctuate"
tokenizer = DistilBertTokenizerFast.from_pretrained(checkpoint)
model = DistilBertForTokenClassification.from_pretrained(checkpoint)
encoder_max_length = 256

#
# Split text to segments of length 200, with overlap 50
#
def split_to_segments(wrds, length, overlap):
    resp = []
    i = 0
    while True:
        wrds_split = wrds[(length * i):((length * (i + 1)) + overlap)]
        if not wrds_split:
            break

        resp_obj = {
            "text": wrds_split,
            "start_idx": length * i,
            "end_idx": (length * (i + 1)) + overlap,
        }

        resp.append(resp_obj)
        i += 1
    return resp


#
# Punctuate wordpieces
#
def punctuate_wordpiece(wordpiece, label):
    if label.startswith('UPPER'):
        wordpiece = wordpiece.upper()
    elif label.startswith('Upper'):
        wordpiece = wordpiece[0].upper() + wordpiece[1:]
    if label[-1] != '_' and label[-1] != wordpiece[-1]:
        wordpiece += label[-1]
    return wordpiece


#
# Punctuate text segments (200 words)
#
def punctuate_segment(wordpieces, word_ids, labels, start_word):
    result = ''
    for idx in range(0, len(wordpieces)):
        if word_ids[idx] == None:
            continue
        if word_ids[idx] < start_word:
            continue
        wordpiece = punctuate_wordpiece(wordpieces[idx][2:] if wordpieces[idx].startswith('##') else wordpieces[idx],
                            labels[idx])
        if idx > 0 and len(result) > 0 and word_ids[idx] != word_ids[idx - 1] and result[-1] != '-':
            result += ' '
        result += wordpiece
    return result


#
# Tokenize, predict, punctuate text segments (200 words)
#
def process_segment(words, tokenizer, model, start_word):

    tokens = tokenizer(words['text'],
                       padding="max_length",
                       # truncation=True,
                       max_length=encoder_max_length,
                       is_split_into_words=True, return_tensors='pt')
    
    with torch.no_grad():
        logits = model(**tokens).logits
    logits = logits.cpu()
    predictions = np.argmax(logits, axis=-1)

    wordpieces = tokens.tokens()
    word_ids = tokens.word_ids()
    id2label = model.config.id2label
    labels = [[id2label[p.item()] for p in prediction] for prediction in predictions][0]

    return punctuate_segment(wordpieces, word_ids, labels, start_word)


#
# Punctuate text of any length
#
def punctuate(text, tokenizer, model):
    text = text.lower()
    text = text.replace('\n', ' ')
    words = text.split(' ')
    
    overlap = 50
    slices = split_to_segments(words, 150, 50)
    
    result = ""
    start_word = 0
    for text in slices:
        corrected = process_segment(text, tokenizer, model, start_word)
        result += corrected + ' '
        start_word = overlap
    return result

#
# Example
#
text = "the atm protein is a single high molecular weight protein predominantly confined to the nucleus of human fibroblasts but is present in both nuclear and microsomal fractions from human lymphoblast cells and peripheral blood lymphocytes atm protein levels and localization remain constant throughout all stages of the cell cycle truncated atm protein was not detected in lymphoblasts from ataxia telangiectasia patients homozygous for mutations leading to premature protein termination exposure of normal human cells to gamma irradiation and the radiomimetic drug neocarzinostatin had no effect on atm protein levels in contrast to a noted rise in p53 levels over the same time interval these findings are consistent with a role for the atm protein in ensuring the fidelity of dna repair and cell cycle regulation following genome damage"
result = punctuate(text, tokenizer, model)
print(result)


"""
Output:
The ATM protein is a single, high-molecular-weight protein predominantly confined to the nucleus of human fibroblasts, but is present in both nuclear and microsomal fractions from human lymphoblast cells and peripheral blood lymphocytes. ATM protein levels and localization remain constant throughout all stages of the cell cycle. Truncated ATM protein was not detected in lymphoblasts from ataxia-telangiectasia-patients homozygous for mutations leading to premature protein termination. Exposure of normal human cells to gamma-irradiation and the radiomimetic drug neocarzinostatin had no effect on ATM protein levels, in contrast to a noted rise in p53 levels over the same time interval. These findings are consistent with a role for the ATM protein in ensuring the fidelity of DNA repair and cell-cycle regulation following genome damage.
"""