SentenceTransformer based on BAAI/bge-small-en-v1.5

Model Details

Model Description

• Model Type: Sentence Transformer
• Base model: BAAI/bge-small-en-v1.5
• Maximum Sequence Length: 512 tokens
• Output Dimensionality: 384 dimensions
• Similarity Function: Cosine Similarity
• Supported Modality: Text

Model Sources

• Documentation: Sentence Transformers Documentation
• Repository: Sentence Transformers on GitHub
• Hugging Face: Sentence Transformers on Hugging Face

Full Model Architecture

SentenceTransformer(
  (0): Transformer({'transformer_task': 'feature-extraction', 'modality_config': {'text': {'method': 'forward', 'method_output_name': 'last_hidden_state'}}, 'module_output_name': 'token_embeddings', 'architecture': 'BertModel'})
  (1): Pooling({'embedding_dimension': 384, 'pooling_mode': 'cls', 'include_prompt': True})
  (2): Normalize({})
)

Usage

Direct Usage (Sentence Transformers)

Training Details

Training Dataset

Unnamed Dataset

• Size: 47,241 training samples
• Columns: sentence_0, sentence_1, and sentence_2
• Approximate statistics based on the first 100 samples:

  	sentence_0	sentence_1	sentence_2
  type	string	string	string
  modality	text	text	text
  details	min: 10 tokens mean: 43.69 tokens max: 67 tokens	min: 80 tokens mean: 113.34 tokens max: 164 tokens	min: 86 tokens mean: 113.38 tokens max: 163 tokens

• Samples:

  sentence_0	sentence_1	sentence_2
  Thioredoxin mediates oxidation-dependent phosphorylation of CRMP2 and growth cone collapse. Semaphorin3A (Sema3A) is a repulsive guidance molecule for axons, which acts by inducing growth cone collaps	Thioredoxin mediates oxidation-dependent phosphorylation of CRMP2 and growth cone collapse. Semaphorin3A (Sema3A) is a repulsive guidance molecule for axons, which acts by inducing growth cone collapse through phosphorylation of CRMP2 (collapsin response mediator protein 2). Here, we show a role for CRMP2 oxidation and thioredoxin (TRX) in the regulation of CRMP2 phosphorylation and growth cone collapse. Sema3A stimulation generated hydrogen peroxide (H2O2) through MICAL (molecule interacting with CasL) and	Differential regulation of actin microfilaments by human MICAL proteins. The Drosophila melanogaster MICAL protein is essential for the neuronal growth cone machinery that functions through plexin- and semaphorin-mediated axonal signaling. Drosophila MICAL is also involved in regulating myofilament organization and synaptic structures, and serves as an actin disassembly factor downstream of plexin-mediated axonal repulsion. In mammalian cells there are three known isoforms, MICAL1, MICAL2 and MICAL3, as wel
  A chromatin remodelling complex involved in transcription and DNA processing. The packaging of the eukaryotic genome in chromatin presents barriers that restrict the access of enzymes that process DNA	Chromatin remodelling at promoters suppresses antisense transcription Chromatin allows the eukaryotic cell to package its DNA efficiently. To understand how chromatin structure is controlled across the Saccharomyces cerevisiae genome, we have investigated the role of the ATP-dependent chromatin remodelling complex Isw2 in positioning nucleosomes. We find that Isw2 functions adjacent to promoter regions where it repositions nucleosomes at the interface between genic and intergenic sequences. Nucleosome repos	High-resolution mapping of open chromatin in the rice genome. Gene expression is controlled by the complex interaction of transcription factors binding to promoters and other regulatory DNA elements. One common characteristic of the genomic regions associated with regulatory proteins is a pronounced sensitivity to DNase I digestion. We generated genome-wide high-resolution maps of DNase I hypersensitive (DH) sites from both seedling and callus tissues of rice (Oryza sativa). Approximately 25% of the DH site
  Attention-deficit hyperactivity disorder: a category or a continuum? Genetic analysis of a large-scale twin study. OBJECTIVE To investigate heritability and continuum versus categorical approaches to	Emotion dysregulation in attention deficit hyperactivity disorder. Although it has long been recognized that many individuals with attention deficit hyperactivity disorder (ADHD) also have difficulties with emotion regulation, no consensus has been reached on how to conceptualize this clinically challenging domain. The authors examine the current literature using both quantitative and qualitative methods. Three key findings emerge. First, emotion dysregulation is prevalent in ADHD throughout the lifespan an	Rare and common variants: twenty arguments Genome-wide association studies have greatly improved our understanding of the genetic basis of disease risk. The fact that they tend not to identify more than a fraction of the specific causal loci has led to divergence of opinion over whether most of the variance is hidden as numerous rare variants of large effect or as common variants of very small effect. Here I review 20 arguments for and against each of these models of the genetic basis of complex traits and

• Loss: MultipleNegativesRankingLoss with these parameters:
  json

  1{ 2 "scale": 20.0, 3 "similarity_fct": "cos_sim", 4 "gather_across_devices": false, 5 "directions": [ 6 "query_to_doc" 7 ], 8 "partition_mode": "joint", 9 "hardness_mode": null, 10 "hardness_strength": 0.0 11}

Training Hyperparameters

Non-Default Hyperparameters

• per_device_train_batch_size: 32
• num_train_epochs: 5
• per_device_eval_batch_size: 32
• multi_dataset_batch_sampler: round_robin

All Hyperparameters

Training Logs

Training Time

• Training: 16.5 minutes

Framework Versions

• Python: 3.13.9
• Sentence Transformers: 5.5.1
• Transformers: 5.9.0
• PyTorch: 2.11.0+cu128
• Accelerate: 1.14.0
• Datasets: 4.8.5
• Tokenizers: 0.22.2

Citation

BibTeX

Sentence Transformers

MultipleNegativesRankingLoss